Daily Peanut Butter Mitigates Age-Related Muscle Decline

By Erik Goldman

A daily serving of peanut butter for 6 months improved muscle power in a cohort of community-dwelling elders. While not a cure-all for age-related muscle decline, it’s an inexpensive and enjoyable way to mitigate risk. (Image: FoodVideoPhoto/Shutterstock)

A few tablespoons of unsweetened peanut butter each day can mitigate some aspects of age-related muscle decline, according to a new study of 120 community-dwelling elders by researchers at the Deakin University School of Exercise & Nutrition Sciences in Geelong, Victoria, Australia.

In general, nuts as a good dietary source of protein, healthy fats, and fiber. Pistachios and peanuts are particularly rich in anti-inflammatory and anti-oxidant phytochemicals that can attenuate chronic inflammation and oxidative stress, which are key drivers of muscle loss.

There’s some evidence from population studies that increased nut consumption is associated with a reduced risk of musculoskeletal and cognitive decline among older people. But until now, the direct impact of daily nut consumption on muscle strength and function had never been evaluated in a randomized intervention trial.  

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HPC Video

TA-65 - Guarding the Genome, Protecting Mitochondria, & Maintaining Telomeres

Written by Erik Goldman

This presentation ‘connects the dots” between telomeres, optimal lifestyle, and health outcomes.

Renowned longevity physician Ron Rothenberg, MD, will reveal how telomeres are beneficially impacted by nutrition, exercise, stress reduction, meditation, yoga, or adversely affected by exposure to radiation, environmental toxins, and poor diet

Dr. Rothenberg will describe the well-known “canonical” effects of telomerase for preventing telomere loss or extending telomere length. There are also significant “non canonical” effects as well, including mitochondrial protection. These may be as important as the nuclear effects.

He’ll also share a comprehensive look at the peer-reviewed medical literature on TA-65–a documented effective telomerase activator derived from a particular species of Astragalus, and it’s key clinical implications.

Key Learning Objectives:

  • Understand the pathophysiology of telomeres and causes of telomere shortening
  • Be able to reinforce the necessity of life style interventions that minimize telomere damage
  • Identify the canonical and non-canonical effects of telomerase
  • Get up to speed on the current peer reviewed literature on effectiveness of a proven telomerase activator
  • Understand the benefits of clinical application of this knowledge

SPONSORED BY T.A. SCIENCES

Ron Rothenberg, MD is among the first 10 physicians certified by the American Board of Anti-Aging and Regenerative Medicine. Throughout his career, he has always forged new paradigms in medicine. After training at Columbia University College of Physicians and Surgeons, he practiced medicine while also studying indigenous healing in the Amazon Basin. He did his Emergency Medicine residency at LA County-USC Medical Center, and then founded the Postgraduate Institute for Primary & Emergency Physicians at UC San Diego’s School of Medicine, where he was a professor of clinical preventive medicine.

Dr. Rothenberg lectures worldwide and has educated 45,000+ physicians with his continuing ed seminars. He is currently medical director of California HealthSpan Institute located on the campus of Scripps Memorial Hospital, Encinitas. Adding to his many publications in anti-aging and preventive medicine, he is the co-author of the book, Hormonal BioIdentity.

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